Why it is necessary to treat genital herpes
Herpes viruses are widely spread in human population, they can dease the host's all organs and systems of the body, causing latent, acute and long-lasting forms of infection.
During the long process of evolution and in connection with the forced coexistence of viruses in the host's body, the herpes viruses have developed various molecular ways to avoid their recognition and elimination of the immune system. According to the results of recent years, various representatives of the Herpesviridae integrate into the genome of infected cells and, by blocking the activity of T-cells and PC cells, protect the infected host's cell from destruction by cytotoxic lymphocytes.
Herpes viruses can also evade the reaction of immune system of the host's body by induction of latency state. This explains the fact of long-lasting infectious process at herpes virus infection. Aiming on the skin nerve, HSV cause microfocus of infection, held by mechanisms of protection, that prevents or reduces recurrence of their symptoms. The state of local immunity influences the development of recurrence. The replication of aimed virus to the skin contributes the inhibition of local immunity.
In latent state the virus persists in the nerve ganglia of people, who have had past infection with HSV, thus it leads to the long-lasting process and the possibility of infection reactivation.
It is necessary to notice, the possible part of Herpes Simplex Virus (HSV), particularly HSV-2 in association with the Human Papilloma Virus, CMV (cytomegalovirus), chlamydias and Mycoplasmas, in development of neoplastic processes in human, such as cervical cancer and prostate cancer (Borisenko K. K., and others, 1997; Isakov V. A. and others, 2006).
HSV-2 can be also a cofactor of carcinogenesis and triggers the development of dysplasia, keeping it in a state of stabilization. Herpes viruses induce the process of atherosclerosis, shown an adverse and sometimes fatal effect of HV on pregnancy and childbirth, the pathology of the fetus and newborn. HVs are a cofactor in HIV infection and AIDS.
According to many authors from 30% to 50% of patients DVC are hidden carriers of Herpes Viruses. Examination of the reproductive secrets of 57 young men with long-lasting inflammatory diseases of internal genital organs by GChMS method (gas chromatography with mass-selective detector) identified at 39 (68,4%) people a specific marker of HSV (herpes simplex virus), at 41 (71,9%) markers of CMVI (cytomegalovirus infection), a combination of viruses presented at 15(26.3%) patients (Y.A. Bazhin, 2002). Having an asymptomatic (subclinical) herpes virus infection is a threat of infection for the sexual partner, in a replicated process of HSV (herpes simplex virus) can infect germ cells, disrupt spermatogenesis, which ultimately appears as a violation of the genetic apparatus. It is also possible the embedding of viral DNA into the genetic apparatus of gametes (E. A. Bragina and others, 2000, 2001). In the survey of 100 men with STIs (sexually transmitted infections) in 1999-2001 the subclinical forms of GG (genital herpes) were identified in 1999 in 12%, in 2000 — 18%, in 2001. - 27% of cases (D. K. Ermolenko and others, 2002).
Ask your gynecologist if he (she) has an experience in treatment of human papillomavirus infection to the complete removal of human papilloma virus from the body?
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